For the clinical manifestations of cirrhosis and portal hypertension, in addition to the following two points, there is the formation of cirrhotic ascites, but ascites is the main clinical manifestation of cirrhosis. What I'm explaining here today are mainly splenomegaly and hypersplenism, as well as the establishment and opening of collateral circulation. These two main manifestations.
1. Splenomegaly and hypersplenism
The spleen swelled due to long-term congestion, mostly mild to moderate swelling. Late splenomegaly is accompanied by decreased red blood cells, white blood cells, and platelets, which is called hypersplenism.
2. Establishment and opening of collateral circulation
Due to the increased portal pressure, the anastomotic branches of the portal vein and vena cava gradually expand, forming a portal-body collateral circulation.
Clinically significant are:
1. Lower esophagus and gastric varices
It is most indicative of portal hypertension, often due to a sudden increase in portal pressure, duodenal gastroesophageal reflux, mechanical damage to hard food, or a sudden increase in intra-abdominal pressure, causing the rupture of varicose veins and causing upper gastrointestinal bleeding, hematemesis, melena and shock And other symptoms;
2. Abdominal wall and periumbilical varicose veins
Curved veins can be seen around the umbilical cord and on the abdominal wall, in the shape of a jellyfish head;
3. Hemorrhoids and varicose veins
Hemorrhoids are formed, which can cause blood in the stool when ruptured.
Most of the theoretical things are relatively abstract and difficult to understand. If you are a clinical medical staff, you can get answers by observing the patient in the ward, but if you are a patient, you should understand the theoretical knowledge and your actual situation. For comparison, patients with liver cirrhosis need to increase their confidence during treatment and cooperate with the doctor's treatment. Only in this way can we alleviate the clinical manifestations of liver cirrhosis.